Search results for "Pancreatic islets"

showing 10 items of 26 documents

GLP-2 as Beneficial Factor in the Glucose Homeostasis in Mice Fed a High Fat Diet

2015

Glucagon like peptide-2 (GLP-2) is a gastrointestinal hormone released in response to dietary nutrients, which acts through a specific receptor, the GLP-2 receptor (GLP-2R). The physiological effects of GLP-2 are multiple, involving also the intestinal adaptation to high fat diet (HFD). In consideration of the well-known relationship between chronic HFD and impaired glucose metabolism, in the present study we examined if the blocking of the GLP-2 signaling by chronic treatment with the GLP-2R antagonist, GLP-2 (3-33), leads to functional consequences in the regulation of glucose metabolism in HFD-fed mice. Compared with animals fed standard diet (STD), mice at the 10th week of HFD showed hy…

endocrine systemmedicine.medical_specialtyPhysiologyPancreatic isletsInsulinmedicine.medical_treatmentdigestive oral and skin physiologyClinical BiochemistryCell BiologyBiologyCarbohydrate metabolismmedicine.diseaseGlucagonEndocrinologymedicine.anatomical_structureInsulin resistanceGlucose Metabolism DisorderInternal medicinemedicineGlucose homeostasisBeta cellhormones hormone substitutes and hormone antagonistsJournal of Cellular Physiology
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Carnosine protects pancreatic beta cells and islets against oxidative stress damage

2018

Abstract Islet transplantation is a valid therapeutic option for type 1 diabetes treatment. However, in this procedure one of the major problems is the oxidative stress produced during pancreatic islet isolation. The aim of our study was to evaluate potential protective effects of L-carnosine and its isomer D-carnosine against oxidative stress. We evaluated the carnosine effect on cell growth, cell death, insulin production, and the main markers of oxidative stress in rat and murine stressed beta cell lines as well as in human pancreatic islets. Both isomers clearly inhibited hydrogen peroxide induced cytotoxicity, with a decrease in intracellular reactive oxygen and nitrogen species, preve…

0301 basic medicineNitrous OxideCarnosineApoptosismedicine.disease_causeBiochemistrychemistry.chemical_compoundMice0302 clinical medicineEndocrinologyInsulin-Secreting CellsInsulin Secretiongeography.geographical_feature_categoryChemistryNitrotyrosineCarnosineDiabetesIsletReactive Nitrogen Speciesmedicine.anatomical_structureBeta cellPancreatic islet transplantationmedicine.medical_specialtyCell SurvivalProtective AgentsCell Line03 medical and health sciencesInternal medicinemedicineAnimalsHumansMolecular BiologyBeta cell lineCell ShapeCell ProliferationSettore MED/04 - Patologia GeneralegeographyPancreatic isletsTranscription Factor RelAHydrogen PeroxideRatsTransplantationOxidative Stress030104 developmental biologyEndocrinologyGlucoseGene Expression RegulationCytoprotectionTyrosinePancreatic islet transplantationReactive Oxygen Species030217 neurology & neurosurgeryOxidative stressBiomarkers
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Glucagon-like peptide-2 treatment improves glucose dysmetabolism in mice fed a high fat diet

2016

Previous studies suggested that endogenous glucagon-like peptide 2 (GLP-2) is dispensable for the regulation of glucose homeostasis under normal conditions, while it can play a beneficial role in obesity conditions. The purpose of the present study was to investigate whether chronic treatment with Gly2-GLP-2, a stable analogue of GLP-2, can have an impact on glycaemic and lipid control in mice fed a high-fat diet (HFD), an animal model of human obesity and insulin resistance. HFD mice were treated once a day with Gly2-GLP-2 for 4 weeks. Body weight, food intake, fasting glucose, intraperitoneal glucose tolerance, insulin-induced glucose clearance, glucose-stimulated insulin secretion, β-cel…

Male0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismDrug Evaluation PreclinicalMicrovesicular SteatosisCarbohydrate metabolismDiet High-FatSettore BIO/09 - FisiologiaRandom Allocation03 medical and health sciencesEndocrinologyInsulin resistanceInternal medicineDiabetes mellitusGlucagon-Like Peptide 2medicineAnimalsGlucose homeostasisObesityPancreasPancreatic islets.Glucose Metabolism Disordersbusiness.industrydigestive oral and skin physiologyInsulin resistancemedicine.diseaseGlucagon-like peptide-2LipidsObesityMice Inbred C57BL030104 developmental biologyEndocrinologyLiverPeptidesbusinessGLP-2Dyslipidemia
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Pancreatic islets from non-heart beating donor pig: Two-layer preservation method in an in vitro porcine model

2011

Purpose Pancreata from non-heart beating donors could represent an unlimited source of islets if their cell viability can be efficiently preserved during the time necessary to process the organs by the use of a better solution of preservation compared to the classic University of Wisconsin solution. The aim of this study was to determine whether it is possible to obtain functioning “alive islets” from non-heart-beating donors by comparing, on a porcine model, the classic “UW ice-store” method with a two-layer cold storage method (TLM) using oxygenated Perfluorocarbons (PFC) and UW. Methods Whole pancreata were harvested from 20 NHBDs female pigs with similar characteristics and preserved fo…

medicine.medical_specialtyAdenosineTime FactorsCell SurvivalSwineTwo-layer method UW solution non-heart beating donor pig pancreas preservationAllopurinolOrgan Preservation SolutionsBiomedical EngineeringIslets of Langerhans TransplantationMedicine (miscellaneous)Cold storageBioengineeringBiologyCold Ischemia TimeCryopreservationBiomaterialsTissue Culture TechniquesIslets of LangerhansRaffinoseSettore BIO/13 - Biologia ApplicataInternal medicinemedicineAnimalsInsulinViaspanViability assayCryopreservationgeographyFluorocarbonsgeography.geographical_feature_categoryPancreatic isletsCold IschemiaGeneral MedicineIsletGlutathioneIn vitroOxygenSettore MED/18 - Chirurgia GeneraleEndocrinologymedicine.anatomical_structureFemaleTissue Preservation
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Diabetic Goto Kakizaki rats as well as type 2 diabetic patients show a decreased diurnal serum melatonin level and an increased pancreatic melatonin-…

2006

There are functional inter-relationships between the beta cells of the endocrine pancreas and the pineal gland, where the synchronizing circadian molecule melatonin originates. The aim of this study was to elucidate a putative interaction between insulin and melatonin in diabetic patients and a diabetic rat model. We analyzed glucose, insulin, and melatonin levels of type 2 patients, as well as type 2 diabetic Goto Kakizaki (GK) rats by radioimmunoassay. Expression of pancreatic melatonin and pineal insulin receptors, as well as arylalkylamine-N-acetyltransferase (AANAT), was determined by real-time reverse transcriptase polymerase chain reaction (RT-PCR). The AANAT enzyme activity was meas…

Maleendocrine systemmedicine.medical_specialtyAANATmedicine.medical_treatmentReceptors MelatoninBiologyArylalkylamine N-AcetyltransferasePineal GlandMelatonin receptorMelatoninPineal glandEndocrinologyInternal medicinemedicineAnimalsHumansInsulinRats WistarPancreasMelatoninPancreatic isletsInsulinReceptor InsulinCircadian RhythmRatsmedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 2Beta cellhormones hormone substitutes and hormone antagonistsEndocrine glandmedicine.drugJournal of Pineal Research
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CD40 activation in human pancreatic islets and ductal cells.

2008

Aims/hypothesis: CD40 expression on non-haematopoietic cells is linked to inflammation. We previously reported that CD40 is expressed on isolated human and non-human primate islets and its activation results in secretion of IL-8, macrophage inflammatory protein 1-beta (MIP-1β) and monocyte chemoattractant protein-1 (MCP-1) through nuclear factor-κB and extracellularly regulated kinases 1/2 pathways. The objective of this study was to identify the pattern of gene expression, and to study viability and functionality affected by CD40-CD40 ligand (CD40L) interaction in human islets. Furthermore, we have studied the CD40-mediated cytokine/chemokine profile in pancreatic ductal cells, as they are…

AdultChemokinemedicine.medical_specialtyDuctal cellsCell SurvivalEndocrinology Diabetes and Metabolismmedicine.medical_treatmentChemokine CXCL1CD40 Chemokines Cytokines Ductal cells Inflammation Insulin Islets of Langerhans Microarray Quantitative RT-PCRCD40 LigandEnzyme-Linked Immunosorbent AssayIslets of LangerhansYoung AdultInternal medicineInternal MedicinemedicineHumansCD40 AntigensMacrophage inflammatory proteinOligonucleotide Array Sequence AnalysisCD40biologySettore BIO/16 - Anatomia UmanaReverse Transcriptase Polymerase Chain ReactionPancreatic isletsPancreatic DuctsMiddle AgedFlow CytometryMolecular biologyCXCL1CXCL2Endocrinologymedicine.anatomical_structureCytokinebiology.proteinCytokinesChemokinesDiabetologia
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Multipotential nestin and Isl-1 positive mesenchymal stem cells isolated from human pancreatic islets.

2006

Mesenchymal cells in the developing pancreas express the neural stem cell marker nestin and the transcription factor islet-1 (Isl-1). Using defined culture conditions we isolated on a single cell basis nestin producing cells from human pancreatic islets. These cells were immortalized with lentiviral vectors coding for telomerase and mBmi. They are positive for Isl-1 and nestin and have the potential to adopt a pancreatic endocrine phenotype with expression of critical transcription factors including Ipf-1, Isl-1, Ngn-3, Pax4, Pax6, Nkx2.2, and Nkx6.1 as well as the islet hormones insulin, glucagon, and somatostatin. In addition, they can be differentiated into human albumin producing cells …

endocrine systemLIM-Homeodomain ProteinsBiophysicsCell Culture TechniquesNerve Tissue ProteinsBiologyBiochemistryNestinIslets of LangerhansIntermediate Filament ProteinsNeurosphereAlbuminsmedicineAdipocytesATP Binding Cassette Transporter Subfamily G Member 2HumansMolecular BiologyStem cell transplantation for articular cartilage repairHomeodomain ProteinsNeuronsOsteoblastsPancreatic isletsMesenchymal stem cellLentivirusNuclear ProteinsCell DifferentiationMesenchymal Stem CellsCell BiologyNestinNeural stem cellNeoplasm Proteinsmedicine.anatomical_structureHomeobox Protein Nkx-2.2Cancer researchPAX4ATP-Binding Cassette TransportersPancreasTranscription FactorsBiochemical and biophysical research communications
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Glucose homeostasis is impaired in mice deficient for the neuropeptide 26RFa (QRFP)

2019

AbstractIntroduction26RFa (QRFP) is a biologically active peptide that has been found to control feeding behaviour by stimulating food intake, and to regulate glucose homeostasis by acting as an incretin. The aim of the present study was thus to investigate the impact of 26RFa gene knockout on the regulation of energy and glucose metabolism.Research design and methods26RFa mutant mice were generated by homologous recombination, in which the entire coding region of prepro-26RFa was replaced by the iCre sequence. Energy and glucose metabolism was evaluated through measurement of complementary parameters. Morphological and physiological alterations of the pancreatic islets were also investigat…

medicine.medical_specialtyPancreatic isletsInsulinmedicine.medical_treatmentQRFPIncretinCarbohydrate metabolismBiologymedicine.diseaseEndocrinologymedicine.anatomical_structureDiabetes mellitusInternal medicinemedicineGlucose homeostasisPancreas
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Overexpression of glucose 6 phosphate dehydrogenase preserves mouse pancreatic beta cells function until late in life.

2021

NAD(P)H donates electrons for reductive biosynthesis and antioxidant defense across all forms of life. Glucose-6- phosphate dehydrogenase (G6PD) is a critical enzyme to provide NADPH. G6PD deficiency is present in more than 400 million people worldwide. This enzymopathy provides protection against malaria but sensitizes cells to oxidative stressors. Oxidative stress has been involved in the pathogenesis of the diabetic complications and several studies have provided evidences of a link between G6PD deficiency and type 2 diabetes (T2D). We hypothesized that a moderate overexpression of G6PD (G6PD-Tg) could protect β-cells from age-associated oxidative stress thus reducing the risk of develop…

0301 basic medicineAgingmedicine.medical_specialtyOxidative phosphorylationType 2 diabetesGlucosephosphate Dehydrogenasemedicine.disease_causeBiochemistry03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicinehemic and lymphatic diseasesPhysiology (medical)Internal medicineDiabetes mellitusInsulin-Secreting Cellsparasitic diseasesNADPHmedicineGlucose-6-phosphate dehydrogenaseAnimalsPancreatic isletsDiabetesWild typenutritional and metabolic diseasesmedicine.diseaseOxidative Stress030104 developmental biologyEndocrinologymedicine.anatomical_structureGlucosephosphate Dehydrogenase DeficiencychemistryDiabetes Mellitus Type 2Oxidative stressPancreas030217 neurology & neurosurgeryOxidative stressFree radical biologymedicine
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Autoimmune Diabetes Recurrence After Pancreas Transplantation: Diagnosis, Management, and Literature Review

2019

[EN] Background: Pancreas transplantation can be a viable treatment option for patients with type 1 diabetes mellitus (T1DM), especially for those who are candidates for kidney transplantation. T1DM may rarely recur after pancreas transplantation, causing the loss of pancreatic graft. The aim of this study was to describe the prevalence of T1DM recurrence after pancreas transplantation in our series. Material/Methods: Eighty-one patients transplanted from 2002 to 2015 were included. Autoantibody testing (GADA and IA-2) was performed before pancreas transplantation and during the follow-up. Results: The series includes 48 males and 33 females, mean age 37.4+5.7 years and mean duration of dia…

AdultGraft RejectionMaleReoperationmedicine.medical_specialtyendocrine system diseasesmedicine.medical_treatment030232 urology & nephrologyAutoimmunity030230 surgeryPancreas transplantationGastroenterologyTECNOLOGIA ELECTRONICA03 medical and health sciences0302 clinical medicineRecurrenceDiabetes mellitusInternal medicinemedicineHumansProspective StudiesKidney transplantationAutoantibodiesOriginal PaperTransplantationType 1 diabetesGlutamate Decarboxylasebusiness.industryPancreatic isletsnutritional and metabolic diseasesImmunosuppressionGeneral Medicinemedicine.diseaseKidney TransplantationTransplantationmedicine.anatomical_structuresurgical procedures operativeDiabetes Mellitus Type 1FemalePancreas TransplantationPancreasbusinessImmunosuppression
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